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2009, ISBN: 1607611775
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2009, ISBN: 1607611775
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2009, ISBN: 1607611775
[EAN: 9781607611776], Neubuch, [SC: 0.0], [PU: Springer-Verlag Gmbh], KREBS (KRANKHEIT) / THERAPIE (DIVERSE); ONKOLOGIE; ONKOLOGIE - RADIOONKOLOGIE; ARZNEI ARZNEIMITTELLEHRE; PHARMAKOLOGI… Mehr…
ISBN: 9781607611776
Much work over the last two decades has firmly established that loss of cell cycle checkpoint regulation, and resultant unabated cellular proliferation, is an inherent characteristic of c… Mehr…
2009
ISBN: 1607611775
Much work over the last two decades has firmly established that loss of cell cycle checkpoint regulation, and resultant unabated cellular proliferation, is an inherent characteristic of c… Mehr…
ISBN: 9781607611776
Much work over the last two decades has firmly established that loss of cell cycle checkpoint regulation, and resultant unabated cellular proliferation, is an inherent characteristic of c… Mehr…
2009, ISBN: 1607611775
2009 Gebundene Ausgabe Krebs (Krankheit) / Therapie (diverse), Pharmakologie, BRCA; DNA; cancerresearch; cancertherapy; Cell; celldeath; leukemia; Lymphoma; ovariancancer; research; Res… Mehr…
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Detailangaben zum Buch - Checkpoint Controls and Targets in Cancer Therapy by Zahid H. Siddik Hardcover | Indigo Chapters
EAN (ISBN-13): 9781607611776
ISBN (ISBN-10): 1607611775
Gebundene Ausgabe
Erscheinungsjahr: 2009
Herausgeber: Zahid H. Siddik
274 Seiten
Gewicht: 0,769 kg
Sprache: eng/Englisch
Buch in der Datenbank seit 2007-12-31T09:44:30+01:00 (Berlin)
Detailseite zuletzt geändert am 2024-03-21T14:17:12+01:00 (Berlin)
ISBN/EAN: 1607611775
ISBN - alternative Schreibweisen:
1-60761-177-5, 978-1-60761-177-6
Alternative Schreibweisen und verwandte Suchbegriffe:
Titel des Buches: checkpoint, therapy, cancer
Daten vom Verlag:
Autor/in: Zahid H. Siddik
Titel: Cancer Drug Discovery and Development; Checkpoint Controls and Targets in Cancer Therapy
Verlag: Humana; Humana Press
274 Seiten
Erscheinungsjahr: 2009-09-07
Totowa; NJ; US
Gedruckt / Hergestellt in Niederlande.
Sprache: Englisch
160,49 € (DE)
164,99 € (AT)
177,00 CHF (CH)
POD
XIV, 274 p. 43 illus., 13 illus. in color.
BB; Hardcover, Softcover / Medizin/Nichtklinische Fächer; Onkologie; Verstehen; BRCA; DNA; cancer; cancer research; cancer therapy; cell; cell death; leukemia; lymphoma; ovarian cancer; research; resistance; tumor; Cancer Biology; Oncology; Pharmacology; Pharmakologie; EA; BC
Circuitry of Checkpoint Response.- Evasion of G1 Checkpoints in Cancer.- Distinct Pathways Involved in S-Phase Checkpoint Control.- Mechanisms of G2 Phase Arrest in DNA Damage-Induced Checkpoint Response.- Centrosomes in Checkpoint Responses.- Interplay of 14-3-3 Family of Proteins with DNA Damage-Regulated Molecules in Checkpoint Control.- Checkpoint Response and the Aetiology of Cancer.- Chromatin Modifications and Orchestration of Checkpoint Response in Cancer.- DNA Damage Response and the Balance Between Cell Survival and Cell Death.- Dysfunction of the RB Retinoblastoma Gene in Cancer.- G1 Phase Cyclins in Cancer Development and Progression.- The BRCA1/2 Pathway Prevents Some Leukemias and Lymphomas in Addition to Breast/Ovarian Cancers: Malignancies that Overcome Checkpoint Controls.- Targeting Checkpoint Response in Cancer Therapy.- Regulation of p53 Activity and Associated Checkpoint Controls.- The Importance of p53 Signaling in the Response of Cells to Checkpoint Inhibitors.- Targeting p21-Dependent Pathways for Cell Death in Cancer Therapy.- p27Kip1 as a Biomarker and Target for Treatment of Cancer.- Targeting Cyclin-Dependent Kinases with Small Molecule Inhibitors.- Chk1 and Chk2 as Checkpoint Targets.- Targeting Cdc25 Phosphatases in Cancer Therapy.Brings together experts, who are defining the field of checkpoints, and as such represents a unique collection of insightful contributions that will serve as an important resource for both the research community and the medical oncologists Since checkpoint regulation is critical in understanding cancer development and cancer therapy, the interest will be diverse and appeal to both basic and translational investigators alike
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