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2012, ISBN: 9781489991782
[ED: Softcover], [PU: Springer / Springer New York / Springer, Berlin], In multicellular organisms the establishment, maintenance, and programmed alterations of cell-type specific gene ex… Mehr…
2014, ISBN: 1489991786
[EAN: 9781489991782], Neubuch, [SC: 0.0], [PU: Springer New York], INFECTIOUS DISEASES, Druck auf Anfrage Neuware - Printed after ordering - In multicellular organisms the establishment, … Mehr…
2014
ISBN: 1489991786
2012 Kartoniert / Broschiert Medizin, allgemein, Immunologie, Mikrobiologie und Virologie, mit Schutzumschlag neu, [PU:Springer New York; Springer US, New York, N.Y.]
ISBN: 9781489991782
Patho-epigenetics Of Disease Books > Health > Medical > Microbiology List_Books
ISBN: 9781489991782
paperback
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Detailangaben zum Buch - Patho-epigenetics Of Disease
EAN (ISBN-13): 9781489991782
ISBN (ISBN-10): 1489991786
Taschenbuch
Erscheinungsjahr: 2014
Herausgeber: Springer-Verlag New York Inc.
Buch in der Datenbank seit 2014-10-10T00:01:56+02:00 (Berlin)
Detailseite zuletzt geändert am 2024-04-04T19:58:28+02:00 (Berlin)
ISBN/EAN: 1489991786
ISBN - alternative Schreibweisen:
1-4899-9178-6, 978-1-4899-9178-2
Alternative Schreibweisen und verwandte Suchbegriffe:
Autor des Buches: min, nill, niller
Titel des Buches: epigenetics
Daten vom Verlag:
Autor/in: Janos Minarovits; Hans Helmut Niller
Titel: Patho-Epigenetics of Disease
Verlag: Springer; Springer US
466 Seiten
Erscheinungsjahr: 2014-06-11
New York; NY; US
Gedruckt / Hergestellt in Niederlande.
Gewicht: 0,730 kg
Sprache: Englisch
160,49 € (DE)
164,99 € (AT)
177,00 CHF (CH)
POD
XIV, 466 p.
BC; Medical Microbiology; Hardcover, Softcover / Medizin/Nichtklinische Fächer; Mikrobiologie und Virologie; Verstehen; infectious diseases; Medicine/Public Health, general; Virology; Infectious Diseases; Immunology; Medical Microbiology; Health Sciences; Virology; Diseases; Immunology; Medizin, allgemein; Mikrobiologie (nicht-medizinisch); Krankheiten und Störungen; Immunologie; BB
In multicellular organisms the establishment, maintenance, and programmed alterations of cell-type specific gene expression patterns are regulated by epigenetic mechanisms. Thus, epigenetic alterations (DNA methylation, DNA associated Polycomb-Trithorax protein complexes, histone modifications) ensure the unique transcriptional activity and phenotypic diversity of diploid cells that carry identical or nearly identical DNA sequences.
Because DNA methyltransferase I (DNMT1) associates with replication foci during S phase and prefers hemimethylated DNA as a substrate, DNMT1 ensures the clonal propagation of cytosine methylation patterns (maintenance methylation). Thus, DNA methylation may provide a memory function by helping progeny cells to “remember” their proper cellular identity.
modification of histone tails.
epigenetic alterations malignant tumors epigenetic dysregulation a wide spectrum of human diseases epigenetic memory via Epigenetic regulatory mechanisms.- Role of DNMT3B Mutations in the Pathogenesis of ICF Syndrome.- Dysfunction of the Methyl-Cpg Binding Protein Mecp2 in Rett Syndrome.- Epigenetic Alterations in Glioblastoma Multiforme.- Aberrant Epigenetic Regulation in Breast Cancer.- The Impact of Epigenetic Alterations on Diagnosis, Prediction and Therapy of Prostate Cancer.- Epigenetic Reprogramming in Lung Carcinomas.- Epigenetic changes in virus associated neoplasms.- Genetic and Epigenetic Determinants of Aggression.- Co-Regulation and Epigenetic Dysregulation in Schizophrenia and Bipolar Disorder.- Disruption of Epigenetic Mechanisms in Autoimmune Syndromes.- Imprinting Disorders.- Epigenetics of cardiovascular diseases.- Microbe-induced epigenetic alterations.In multicellular organisms the establishment, maintenance, and programmed alterations of cell-type specific gene expression patterns are regulated by epigenetic mechanisms. Thus, epigenetic alterations (DNA methylation, DNA associated Polycomb-Trithorax protein complexes, histone modifications) ensure the unique transcriptional activity and phenotypic diversity of diploid cells that carry identical or nearly identical DNA sequences.
Because DNA methyltransferase I (DNMT1) associates with replication foci during S phase and prefers hemimethylated DNA as a substrate, DNMT1 ensures the clonal propagation of cytosine methylation patterns (maintenance methylation). Thus, DNA methylation may provide a memory function by helping progeny cells to “remember” their proper cellular identity.
modification of histone tails.
epigenetic alterations malignant tumors epigenetic dysregulation a wide spectrum of human diseases epigenetic memory viaPatho-Epigenetics is a new discipline, and it’s covered here in detail
Written by experts in this exciting new field
Brings together recent results of epigenetic research with special attention to disease development
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